Your trabecular processes comprise a mineral-rich endoskeleton supporting the weight of your meat mass. Calcium is deposited along the trajectory of each filiform arm-sheath by virtue of nutrient-rich Vitamin D projectiles penetrating the dentritic membrane through semi permeable bone holes.

Once past the articular sheath, these projectiles explode with monoclimactic ganglyocytes that encode an activating enzyme to cleave to your archtypal homunculus.  This allows for greater mass and stability within the trabecular architecture, leading to heterogeneous quality-of-life endpoint expectations.

Studies in osteoporotic zebras being treated for external lumbar pre-diabetes suggest that intradermal administration of 60 mg bi-weekly of an ambivalent monocyte interrupts a step in the mitochondrial cascade that plunges normal trabeculae into undifferentiated stasis.  p value = 4.  This study shows promise for treatment of unstable trabeculae vulnerable to sudden calcium attack by freefloating X variables.  More study is needed.